Kto

Kto Rechtschreibung

KTO ist ein französischsprachiger katholischer Fernsehsender. Es wird in Frankreich, Belgien und der Schweiz sowie in frankophonen Ländern im Nahen Osten und in Afrika südlich der Sahara ausgestrahlt. Kto. (Deutsch)Bearbeiten · AbkürzungBearbeiten. Bedeutungen: [1] Konto. Beispiele: [1] Kto. aufgelöst und gelöscht. ÜbersetzungenBearbeiten · Englisch: [​1]. Übersetzung Polnisch-Deutsch für kto im PONS Online-Wörterbuch nachschlagen! Gratis Vokabeltrainer, Verbtabellen, Aussprachefunktion. Kto. Kontonummer. kto: Bedeutung. kto, Konto. kto, kontonumer. Kto. beim Online Wöpinkaminka.nl: ✓ Bedeutung, ✓ Definition, ✓ Rechtschreibung, ✓ Anwendungsbeispiele.

Kto

Kto. Kontonummer. kto: Bedeutung. kto, Konto. kto, kontonumer. Kto. beim Online Wöpinkaminka.nl: ✓ Bedeutung, ✓ Definition, ✓ Rechtschreibung, ✓ Anwendungsbeispiele. KTO – die Schule mit dem demokratischen Leitbild und dem musischen Profil. „​Die vielen Projekte am Tag der offenen Tür haben mir gut gefallen. Wir gingen in​.

BESTE SPIELOTHEK IN SEISELITZ FINDEN Kto Er sich leicht Гberkaufen und Casinos Kto es so, dass.

Kto Beste Spielothek in Bevers finden
Otoslotto Über den Rechtschreibduden. Www Spiele Umsonst De aus dem Etruskischen. Boarders have been pilgrimaging to the legendary KTOKaunertal Opening, every year in October for the past 33 years. KTO Rosomak vehicles including Deuces Wild medevac in Afghanistan since Sie sind öfter hier?
HOCUS Online Spielsucht Gambling
GELD VON PAYPAL AUF KONTO ГЈBERWEISEN DAUER 260

Son , Mart , Ilk. Yap , Son , Lad. Yap , Son , Mart. Kebun Tanaman Obat. Indonesia , Technology , Analytics.

Keep Tube Open. Kickapoo Tribe of Oklahoma. Klant Tevredenheids Onderzoek. Next Suggest to this list. Related acronyms and abbreviations Abbr.

Meaning CPU. Central Processing Unit. Combat Logistics Support Squadron. Makine Kimya Endüstrisi. Eugene Island. Gulf Coast Countries.

Foods within each of these four groups may be freely substituted to allow for variation without needing to recalculate portion sizes. For example, cooked broccoli, Brussels sprouts, cauliflower, and green beans are all equivalent.

Fresh, canned, or frozen foods are equivalent, but raw and cooked vegetables differ, and processed foods are an additional complication.

The child must eat the whole meal and cannot have extra portions; any snacks must be incorporated into the meal plan. A small amount of MCT oil may be used to help with constipation or to increase ketosis.

The classic ketogenic diet is not a balanced diet and only contains tiny portions of fresh fruit and vegetables, fortified cereals, and calcium-rich foods.

In particular, the B vitamins , calcium , and vitamin D must be artificially supplemented. This is achieved by taking two sugar-free supplements designed for the patient's age: a multivitamin with minerals and calcium with vitamin D.

Normal dietary fat contains mostly long-chain triglycerides LCTs. Medium-chain triglycerides MCTs are more ketogenic than LCTs because they generate more ketones per unit of energy when metabolised.

Their use allows for a diet with a lower proportion of fat and a greater proportion of protein and carbohydrate, [18] leading to more food choices and larger portion sizes.

The classical and modified MCT ketogenic diets are equally effective and differences in tolerability are not statistically significant.

First reported in , the idea of using a form of the Atkins diet to treat epilepsy came about after parents and patients discovered that the induction phase of the Atkins diet controlled seizures.

The ketogenic diet team at Johns Hopkins Hospital modified the Atkins diet by removing the aim of achieving weight loss, extending the induction phase indefinitely, and specifically encouraging fat consumption.

Compared with the ketogenic diet, the modified Atkins diet MAD places no limit on calories or protein, and the lower overall ketogenic ratio about does not need to be consistently maintained by all meals of the day.

The MAD does not begin with a fast or with a stay in hospital and requires less dietitian support than the ketogenic diet.

Like the ketogenic diet, the MAD requires vitamin and mineral supplements and children are carefully and periodically monitored at outpatient clinics.

The low glycemic index treatment LGIT [51] is an attempt to achieve the stable blood glucose levels seen in children on the classic ketogenic diet while using a much less restrictive regimen.

The hypothesis is that stable blood glucose may be one of the mechanisms of action involved in the ketogenic diet, [46] which occurs because the absorption of the limited carbohydrates is slowed by the high fat content.

Like the modified Atkins diet, the LGIT is initiated and maintained at outpatient clinics and does not require precise weighing of food or intensive dietitian support.

Both are offered at most centres that run ketogenic diet programmes, and in some centres they are often the primary dietary therapy for adolescents.

The data coming from one centre's experience with 76 children up to the year also indicate fewer side effects than the ketogenic diet and that it is better tolerated, with more palatable meals.

Infants and patients fed via a gastrostomy tube can also be given a ketogenic diet. Parents make up a prescribed powdered formula, such as KetoCal, into a liquid feed.

It is used to administer the ratio classic ketogenic diet in children over one year. The formula is available in both and ratios, either unflavoured or in an artificially sweetened vanilla flavour and is suitable for tube or oral feeding.

There are theoretically no restrictions on where the ketogenic diet might be used, and it can cost less than modern anticonvulsants.

However, fasting and dietary changes are affected by religious and cultural issues. A culture where food is often prepared by grandparents or hired help means more people must be educated about the diet.

When families dine together, sharing the same meal, it can be difficult to separate the child's meal. In many countries, food labelling is not mandatory, so calculating macronutrients such as fat, protein and carbohydrates can be difficult.

In some countries, it may be hard to find sugar-free forms of medicines and supplements, to purchase an accurate electronic scale, or to afford MCT oils.

In Asia, the traditional diet includes rice and noodles as the main source of energy, making their elimination difficult. Therefore, the MCT-oil form of the diet, which allows more carbohydrate, has proved useful.

In India, religious beliefs commonly affect the diet: some patients are vegetarians and will not eat root vegetables and will try to avoid beef.

The Indian ketogenic diet is started without a fast due to cultural opposition towards fasting in children.

The low-fat, high-carbohydrate nature of the normal Indian and Asian diet means that their ketogenic diets typically have a lower ketogenic ratio than in America and Europe.

However, they appear to be just as effective. In many developing countries, the ketogenic diet is expensive because dairy fats and meat are more expensive than grain, fruit, and vegetables.

The modified Atkins diet has been proposed as a lower-cost alternative for those countries; the slightly more expensive food bill can be offset by a reduction in pharmaceutical costs if the diet is successful.

The modified Atkins diet is less complex to explain and prepare and requires less support from a dietitian. The brain is composed of a network of neurons that transmit signals by propagating nerve impulses.

The propagation of this impulse from one neuron to another is typically controlled by neurotransmitters , though there are also electrical pathways between some neurons.

A neuron that releases inhibitory neurotransmitters from its terminals is called an inhibitory neuron, while one that releases excitatory neurotransmitters is an excitatory neuron.

When the normal balance between inhibition and excitation is significantly disrupted in all or part of the brain, a seizure can occur.

The GABA system is an important target for anticonvulsant drugs, since seizures may be discouraged by increasing GABA synthesis, decreasing its breakdown, or enhancing its effect on neurons.

The nerve impulse is characterised by a great influx of sodium ions through channels in the neuron's cell membrane followed by an efflux of potassium ions through other channels.

The neuron is unable to fire again for a short time known as the refractory period , which is mediated by another potassium channel. The flow through these ion channels is governed by a "gate" which is opened by either a voltage change or a chemical messenger known as a ligand such as a neurotransmitter.

These channels are another target for anticonvulsant drugs. There are many ways in which epilepsy occurs. Examples of pathological physiology include: unusual excitatory connections within the neuronal network of the brain; abnormal neuron structure leading to altered current flow; decreased inhibitory neurotransmitter synthesis; ineffective receptors for inhibitory neurotransmitters; insufficient breakdown of excitatory neurotransmitters leading to excess; immature synapse development; and impaired function of ionic channels.

Although many hypotheses have been put forward to explain how the ketogenic diet works, it remains a mystery. Disproven hypotheses include systemic acidosis high levels of acid in the blood , electrolyte changes and hypoglycaemia low blood glucose.

The lack of understanding in this area is similar to the situation with many anticonvulsant drugs. On the ketogenic diet, carbohydrates are restricted and so cannot provide for all the metabolic needs of the body.

Instead, fatty acids are used as the major source of fuel. These are used through fatty-acid oxidation in the cell's mitochondria the energy-producing parts of the cell.

Humans can convert some amino acids into glucose by a process called gluconeogenesis , but cannot do this by using fatty acids.

This could pose a problem for the brain, since it is normally fuelled solely by glucose, and most fatty acids do not cross the blood—brain barrier.

These ketone bodies enter the brain and partially substitute for blood glucose as a source of energy. The ketone bodies are possibly anticonvulsant; in animal models , acetoacetate and acetone protect against seizures.

The ketogenic diet results in adaptive changes to brain energy metabolism that increase the energy reserves; ketone bodies are a more efficient fuel than glucose, and the number of mitochondria is increased.

This may help the neurons to remain stable in the face of increased energy demand during a seizure, and may confer a neuroprotective effect.

The ketogenic diet has been studied in at least 14 rodent animal models of seizures. It is protective in many of these models and has a different protection profile than any known anticonvulsant.

Conversely, fenofibrate , not used clinically as an antiepileptic, exhibits experimental anticonvulsant properties in adult rats comparable to the ketogenic diet.

Anticonvulsants suppress epileptic seizures, but they neither cure nor prevent the development of seizure susceptibility. The development of epilepsy epileptogenesis is a process that is poorly understood.

A few anticonvulsants valproate , levetiracetam and benzodiazepines have shown antiepileptogenic properties in animal models of epileptogenesis.

However, no anticonvulsant has ever achieved this in a clinical trial in humans. The ketogenic diet has been found to have antiepileptogenic properties in rats.

The ketogenic diet has been studied for potential therapeutic use in various neurological disorders other than epilepsy: Alzheimer's disease AD , amyotrophic lateral sclerosis ALS , autism , headache, neurotrauma , pain, Parkinson's disease PD and sleep disorders.

Because some cancer cells are inefficient in processing ketone bodies for energy , the ketogenic diet has also been suggested as a treatment for cancer.

The clinical studies in humans are typically very small, with some providing weak evidence for an anti-tumour effect, particularly for glioblastoma , but in other cancers and studies, no anti-tumour effect was seen.

Taken together, results from preclinical studies, albeit sometimes contradictory, tend to support an anti-tumor effect rather than a pro-tumor effect of the KD for most solid cancers.

The evidence of benefit for these conditions has not reached the level where clinical recommendations can be made. From Wikipedia, the free encyclopedia.

This is the latest accepted revision , reviewed on 3 August This article is about a dietary therapy for epilepsy. For information on ketogenic diets as a lifestyle choice or for weight loss, see Low-carbohydrate diet.

High-fat, adequate-protein, low-carbohydrate diet. Ketone bodies. Erasistrateos Romae degentes , c. The ketogenic diet: one decade later.

Ketogenic diets for drug-resistant epilepsy. Cochrane Database Syst Rev. Dietary therapies for epilepsy. Biomed J. Medium-chain triglyceride MCT ketogenic therapy.

An overview of the ketogenic diet for pediatric epilepsy. Nutr Clin Pract. Ketogenic diets, mitochondria, and neurological diseases. J Lipid Res. An introduction to seizures and epilepsy.

Epilepsy and the ketogenic diet. Totowa: Humana Press; The global burden and stigma of epilepsy. Epilepsy Behav. Safety and tolerability of the ketogenic diet used for the treatment of refractory childhood epilepsy: a systematic review of published prospective studies.

World J Pediatr. History and origin of the ketogenic diet PDF. The falling sickness: a history of epilepsy from the Greeks to the beginnings of modern neurology.

Baltimore: Johns Hopkins University Press; Rev Ther Med-Chirurg. The use of diet in the treatment of epilepsy. Do ketogenic diets work for adults with epilepsy?

Cited 24 October Medium-chain triglycerides as a therapy for intractable childhood epilepsy. The ketogenic diet for the treatment of childhood epilepsy: a randomised controlled trial.

Lancet Neurol. A multicenter study of the efficacy of the ketogenic diet. Arch Neurol. Ketogenic diets: an update for child neurologists.

J Child Neurol. Optimal clinical management of children receiving dietary therapies for epilepsy: Updated recommendations of the International Ketogenic Diet Study Group.

Epilepsia Open. The ketogenic diet all grown up-Ketogenic diet therapies for adults. Epilepsy Res. Who decides the list of beneficiary countries and on the basis of which criteria?

And there arose a reasoning among them, which of them was the greatest. Taking the example of the antiviral drugs: who has, and who does not have, these?

It is not for the European Union to say who must or who must not govern these countries. Greece is going to need bailing out - but by whom?

It is essential to understand what is needed, by whom and when. Synonyms Synonyms Polish for "kto":.

Das Dudenkorpus. Hallo Welt. Über den Kto. Ungarisch Wörterbücher. Beispielsätze für kto kto tam? Möchten Sie ein Wort, eine Phrase oder eine Übersetzung Android App Deutsch As described there, your name or advertisement will appear Beste Spielothek in Sieringhoek finden the programme, in a size proportional to your contribution. Das Wort des Tages. Auf Grund von schlechten Erfahrungen müssen wir leider auch auf Vorkasse bestehen kto Nr. Wie kommt ein Wort in Feiertage Nrw 18 Duden? Wann kann der Bindestrich gebraucht werden? Kto

Kto Video

The Evolution of Mobile Quarterbacks Die Wörter mit den meisten aufeinanderfolgenden Vokalen. Wort Advcash Deutsch Unwort des Jahres in Deutschland. Registrieren Einloggen. Hallo Welt. Otherwise your message will be regarded as spam. Konrad Duden. Über die Duden-Sprachberatung. KTO – die Schule mit dem demokratischen Leitbild und dem musischen Profil. „​Die vielen Projekte am Tag der offenen Tür haben mir gut gefallen. Wir gingen in​. KTO Télévision Catholique, Malakoff. Gefällt Mal · Personen sprechen darüber · waren hier. Regardez KTO. Abkürzungen - Kto. (5 ähnliche Treffer). Kurzform. Langform. Spr. Üb. Kat. Unterkateg. Typ. Abkürzung: AktO. Aktienordnung. Fremdwort. Recht. Ordnung. Kto. Kurz für: Konto. Weitere Vorteile gratis testen. Sie sind öfter hier? Dann sollten Sie einen. Übersetzung im Kontext von „Kto“ in Deutsch-Englisch von Reverso Context: In Valerij Prijomichovs Kto, esli ne mij | Wer wenn nicht wir ist die russische Jugend​. Kto The channel was founded in by Jean-Marie Lustigerwho served as the Archbishop of Paris from to Gisela Walsken overview of the ketogenic diet for pediatric epilepsy. Conklin conjectured that epileptic seizures were caused when a toxin, secreted from the Peyer's patches in the intestines, was discharged into the bloodstream. The ketogenic diet team Kto Johns Hopkins Hospital modified the Atkins Kurs Dash by removing the aim of achieving weight loss, extending the induction Poker AГџistent indefinitely, and specifically encouraging fat consumption. A Cochrane systematic review in found and analysed eleven randomized controlled trials of ketogenic diet in people with epilepsy for whom drugs failed Geschehen Sich 8 Buchstaben control their seizures. A study with an intent-to-treat prospective design was published in by a team from the Johns Hopkins Hospital [22] and followed-up by a report published in Concerns over child health and growth led to a relaxation of Beste Spielothek in Altenricht finden diet's restrictions. However, many patients Kto up the diet, for various reasons, and the quality of evidence was inferior to studies on children. Diet food. Retrieved March 28,

Kto Video

Comparing Former NFL Stars to their Sons

It is not for the European Union to say who must or who must not govern these countries. Greece is going to need bailing out - but by whom?

It is essential to understand what is needed, by whom and when. Synonyms Synonyms Polish for "kto":. Context sentences Context sentences for "kto" in English These sentences come from external sources and may not be accurate.

More by bab. Living abroad Tips and Hacks for Living Abroad Everything you need to know about life in a foreign country. Highly active children or those with muscle spasticity require more food energy than this; immobile children require less.

The ketogenic ratio of the diet compares the weight of fat to the combined weight of carbohydrate and protein.

The quantity of fat in the diet can be calculated from the overall energy requirements and the chosen ketogenic ratio.

Lastly, the amount of carbohydrate is set according to what allowance is left while maintaining the chosen ratio. Any carbohydrate in medications or supplements must be subtracted from this allowance.

The total daily amount of fat, protein, and carbohydrate is then evenly divided across the meals. A computer program such as KetoCalculator may be used to help generate recipes.

Only low-carbohydrate fruits and vegetables are allowed, which excludes bananas, potatoes, peas, and corn. Suitable fruits are divided into two groups based on the amount of carbohydrate they contain, and vegetables are similarly divided into two groups.

Foods within each of these four groups may be freely substituted to allow for variation without needing to recalculate portion sizes.

For example, cooked broccoli, Brussels sprouts, cauliflower, and green beans are all equivalent. Fresh, canned, or frozen foods are equivalent, but raw and cooked vegetables differ, and processed foods are an additional complication.

The child must eat the whole meal and cannot have extra portions; any snacks must be incorporated into the meal plan. A small amount of MCT oil may be used to help with constipation or to increase ketosis.

The classic ketogenic diet is not a balanced diet and only contains tiny portions of fresh fruit and vegetables, fortified cereals, and calcium-rich foods.

In particular, the B vitamins , calcium , and vitamin D must be artificially supplemented. This is achieved by taking two sugar-free supplements designed for the patient's age: a multivitamin with minerals and calcium with vitamin D.

Normal dietary fat contains mostly long-chain triglycerides LCTs. Medium-chain triglycerides MCTs are more ketogenic than LCTs because they generate more ketones per unit of energy when metabolised.

Their use allows for a diet with a lower proportion of fat and a greater proportion of protein and carbohydrate, [18] leading to more food choices and larger portion sizes.

The classical and modified MCT ketogenic diets are equally effective and differences in tolerability are not statistically significant.

First reported in , the idea of using a form of the Atkins diet to treat epilepsy came about after parents and patients discovered that the induction phase of the Atkins diet controlled seizures.

The ketogenic diet team at Johns Hopkins Hospital modified the Atkins diet by removing the aim of achieving weight loss, extending the induction phase indefinitely, and specifically encouraging fat consumption.

Compared with the ketogenic diet, the modified Atkins diet MAD places no limit on calories or protein, and the lower overall ketogenic ratio about does not need to be consistently maintained by all meals of the day.

The MAD does not begin with a fast or with a stay in hospital and requires less dietitian support than the ketogenic diet. Like the ketogenic diet, the MAD requires vitamin and mineral supplements and children are carefully and periodically monitored at outpatient clinics.

The low glycemic index treatment LGIT [51] is an attempt to achieve the stable blood glucose levels seen in children on the classic ketogenic diet while using a much less restrictive regimen.

The hypothesis is that stable blood glucose may be one of the mechanisms of action involved in the ketogenic diet, [46] which occurs because the absorption of the limited carbohydrates is slowed by the high fat content.

Like the modified Atkins diet, the LGIT is initiated and maintained at outpatient clinics and does not require precise weighing of food or intensive dietitian support.

Both are offered at most centres that run ketogenic diet programmes, and in some centres they are often the primary dietary therapy for adolescents.

The data coming from one centre's experience with 76 children up to the year also indicate fewer side effects than the ketogenic diet and that it is better tolerated, with more palatable meals.

Infants and patients fed via a gastrostomy tube can also be given a ketogenic diet. Parents make up a prescribed powdered formula, such as KetoCal, into a liquid feed.

It is used to administer the ratio classic ketogenic diet in children over one year. The formula is available in both and ratios, either unflavoured or in an artificially sweetened vanilla flavour and is suitable for tube or oral feeding.

There are theoretically no restrictions on where the ketogenic diet might be used, and it can cost less than modern anticonvulsants. However, fasting and dietary changes are affected by religious and cultural issues.

A culture where food is often prepared by grandparents or hired help means more people must be educated about the diet. When families dine together, sharing the same meal, it can be difficult to separate the child's meal.

In many countries, food labelling is not mandatory, so calculating macronutrients such as fat, protein and carbohydrates can be difficult.

In some countries, it may be hard to find sugar-free forms of medicines and supplements, to purchase an accurate electronic scale, or to afford MCT oils.

In Asia, the traditional diet includes rice and noodles as the main source of energy, making their elimination difficult.

Therefore, the MCT-oil form of the diet, which allows more carbohydrate, has proved useful. In India, religious beliefs commonly affect the diet: some patients are vegetarians and will not eat root vegetables and will try to avoid beef.

The Indian ketogenic diet is started without a fast due to cultural opposition towards fasting in children.

The low-fat, high-carbohydrate nature of the normal Indian and Asian diet means that their ketogenic diets typically have a lower ketogenic ratio than in America and Europe.

However, they appear to be just as effective. In many developing countries, the ketogenic diet is expensive because dairy fats and meat are more expensive than grain, fruit, and vegetables.

The modified Atkins diet has been proposed as a lower-cost alternative for those countries; the slightly more expensive food bill can be offset by a reduction in pharmaceutical costs if the diet is successful.

The modified Atkins diet is less complex to explain and prepare and requires less support from a dietitian. The brain is composed of a network of neurons that transmit signals by propagating nerve impulses.

The propagation of this impulse from one neuron to another is typically controlled by neurotransmitters , though there are also electrical pathways between some neurons.

A neuron that releases inhibitory neurotransmitters from its terminals is called an inhibitory neuron, while one that releases excitatory neurotransmitters is an excitatory neuron.

When the normal balance between inhibition and excitation is significantly disrupted in all or part of the brain, a seizure can occur.

The GABA system is an important target for anticonvulsant drugs, since seizures may be discouraged by increasing GABA synthesis, decreasing its breakdown, or enhancing its effect on neurons.

The nerve impulse is characterised by a great influx of sodium ions through channels in the neuron's cell membrane followed by an efflux of potassium ions through other channels.

The neuron is unable to fire again for a short time known as the refractory period , which is mediated by another potassium channel.

The flow through these ion channels is governed by a "gate" which is opened by either a voltage change or a chemical messenger known as a ligand such as a neurotransmitter.

These channels are another target for anticonvulsant drugs. There are many ways in which epilepsy occurs. Examples of pathological physiology include: unusual excitatory connections within the neuronal network of the brain; abnormal neuron structure leading to altered current flow; decreased inhibitory neurotransmitter synthesis; ineffective receptors for inhibitory neurotransmitters; insufficient breakdown of excitatory neurotransmitters leading to excess; immature synapse development; and impaired function of ionic channels.

Although many hypotheses have been put forward to explain how the ketogenic diet works, it remains a mystery. Disproven hypotheses include systemic acidosis high levels of acid in the blood , electrolyte changes and hypoglycaemia low blood glucose.

The lack of understanding in this area is similar to the situation with many anticonvulsant drugs.

On the ketogenic diet, carbohydrates are restricted and so cannot provide for all the metabolic needs of the body.

Instead, fatty acids are used as the major source of fuel. These are used through fatty-acid oxidation in the cell's mitochondria the energy-producing parts of the cell.

Humans can convert some amino acids into glucose by a process called gluconeogenesis , but cannot do this by using fatty acids.

This could pose a problem for the brain, since it is normally fuelled solely by glucose, and most fatty acids do not cross the blood—brain barrier.

These ketone bodies enter the brain and partially substitute for blood glucose as a source of energy.

The ketone bodies are possibly anticonvulsant; in animal models , acetoacetate and acetone protect against seizures.

The ketogenic diet results in adaptive changes to brain energy metabolism that increase the energy reserves; ketone bodies are a more efficient fuel than glucose, and the number of mitochondria is increased.

This may help the neurons to remain stable in the face of increased energy demand during a seizure, and may confer a neuroprotective effect.

The ketogenic diet has been studied in at least 14 rodent animal models of seizures. It is protective in many of these models and has a different protection profile than any known anticonvulsant.

Conversely, fenofibrate , not used clinically as an antiepileptic, exhibits experimental anticonvulsant properties in adult rats comparable to the ketogenic diet.

Anticonvulsants suppress epileptic seizures, but they neither cure nor prevent the development of seizure susceptibility. The development of epilepsy epileptogenesis is a process that is poorly understood.

A few anticonvulsants valproate , levetiracetam and benzodiazepines have shown antiepileptogenic properties in animal models of epileptogenesis.

However, no anticonvulsant has ever achieved this in a clinical trial in humans. The ketogenic diet has been found to have antiepileptogenic properties in rats.

The ketogenic diet has been studied for potential therapeutic use in various neurological disorders other than epilepsy: Alzheimer's disease AD , amyotrophic lateral sclerosis ALS , autism , headache, neurotrauma , pain, Parkinson's disease PD and sleep disorders.

Because some cancer cells are inefficient in processing ketone bodies for energy , the ketogenic diet has also been suggested as a treatment for cancer.

The clinical studies in humans are typically very small, with some providing weak evidence for an anti-tumour effect, particularly for glioblastoma , but in other cancers and studies, no anti-tumour effect was seen.

Taken together, results from preclinical studies, albeit sometimes contradictory, tend to support an anti-tumor effect rather than a pro-tumor effect of the KD for most solid cancers.

The evidence of benefit for these conditions has not reached the level where clinical recommendations can be made.

From Wikipedia, the free encyclopedia. This is the latest accepted revision , reviewed on 3 August This article is about a dietary therapy for epilepsy.

For information on ketogenic diets as a lifestyle choice or for weight loss, see Low-carbohydrate diet. High-fat, adequate-protein, low-carbohydrate diet.

Ketone bodies. Erasistrateos Romae degentes , c. The ketogenic diet: one decade later. Ketogenic diets for drug-resistant epilepsy. Cochrane Database Syst Rev.

Dietary therapies for epilepsy. Biomed J. Medium-chain triglyceride MCT ketogenic therapy. An overview of the ketogenic diet for pediatric epilepsy.

Nutr Clin Pract. Ketogenic diets, mitochondria, and neurological diseases. J Lipid Res. An introduction to seizures and epilepsy.

Epilepsy and the ketogenic diet. Totowa: Humana Press; The global burden and stigma of epilepsy. Epilepsy Behav.

Safety and tolerability of the ketogenic diet used for the treatment of refractory childhood epilepsy: a systematic review of published prospective studies.

World J Pediatr. History and origin of the ketogenic diet PDF. The falling sickness: a history of epilepsy from the Greeks to the beginnings of modern neurology.

Baltimore: Johns Hopkins University Press; Rev Ther Med-Chirurg. The use of diet in the treatment of epilepsy. Do ketogenic diets work for adults with epilepsy?

Cited 24 October Medium-chain triglycerides as a therapy for intractable childhood epilepsy. The ketogenic diet for the treatment of childhood epilepsy: a randomised controlled trial.

Retrieved March 28, Valeurs actuelles in French. March 9, Archived from the original on April 28, France June 14, Retrieved June 14, KTO television catholique in French.

June 15,